Shigeaki Kato, the former University of Tokyo endocrinology researcher who resigned in 2012 and has retracted at least ten papers, by our count, has five more retractions.
Here are the papers, all in the Journal of Biological Chemistry (JBC):
- Positive and negative modulation of vitamin D receptor function by transforming growth factor-β signaling through Smad proteins (cited 84 times, according to Thomson Scientific’s Web of Knowledge)
- Ligand type-specific interactions of peroxisome proliferator-activated receptor γ with transcriptional coactivators (cited 163 times)
- The tamoxifen-responsive estrogen receptor α mutant D351Y shows reduced tamoxifen-dependent interaction with corepressor complexes (cited 49 times)
- An hGCN5/TRRAP histone acetyltransferase complex co-activates BRCA1 transactivation function through histone modification (cited 25 times)
- Phosphorylation of Williams syndrome transcription factor by MAPK induces a switching between two distinct chromatin remodeling complexes (cited 15 times)
Despite the investigations, unfortunately, as we have often sees at the JBC, the notices are scant, all saying simply:
This article has been withdrawn by the authors.
This isn’t likely the last you’ll hear of Kato on Retraction Watch. Five of his papers are subject to an expression of concern, and a University of Tokyo panel recommended that 43 of his papers be retracted.